Worn out from horseback riding, the 10-year-old sat quietly on a recent summer afternoon, smiling, amused by his best friend, Harrison Spiers.
Harrison, for his part, was hosting a one-man yak-fest, and his topic was one any 10-year-old boy, or former 10-year-old boy, should relate to: baseball.
Neither boy, though, volunteered comment about what will surely be the most memorable event of their activity-packed summer: the trip to Germany they’re about to take. Asked about it, the boys shrug it off. No big deal. We’ve flown in airplanes before.
No big deal, except that this trip isn’t about a relaxing summer getaway
New University at Buffalo research demonstrates how defects in an important neurological pathway in early development may be responsible for the onset of schizophrenia later in life.
The UB findings, published in Schizophrenia Research (paper at http://bit.ly/Wq1i41), test the hypothesis in a new mouse model of schizophrenia that demonstrates how gestational brain changes cause behavioral problems later in life – just like the human disease.
Partial funding for the research came from New York Stem Cell Science (NYSTEM).
The genomic pathway, called the Integrative Nuclear FGFR 1 Signaling (INFS), is a central intersection point for multiple pathways of as many as 160